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The Journal of Basic and Applied Zoology
Fig. 4 | The Journal of Basic and Applied Zoology

Fig. 4

From: Ameliorative effect of bee venom and its extracted bradykinin-potentiating factor on neurological alteration induced by acrylamide and chips administration

Fig. 4

Photomicrographs of stained sections of the hippocampus showing the anatomy (a) and the small pyramidal cells in the CA1 zone of the control group (A1). Inflammation cells (white arrows) and cell vacuolation (black arrow) of the CA1 zone in the acrylamide-administrated group for 60 days (d) are shown compared to the control (A1) and acrylamide administration for 30 and 45 days (b, c), respectively. Cell vacuolation of the CA1 cell zone in the chips feeding group (arrows, g) is shown at 60 days compared to both 30 and 45 days (d, f) of the chips feeding group. The BV- (h) and BPF- (i) treated groups for 60 days are similar to the control group. The acrylamide-administrated groups treated either with BV (j) or with BPF (k) showed improvement as compared with the acrylamide-administrated group for 60 days. The chips feeding groups treated either with BV (l) or with BPF (m) showed improvement as compared with the chips feeding group for 60 days. H&E stain; scale bar, A is 50 μm; A1–M is 10 μm

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