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The Journal of Basic and Applied Zoology
Fig. 5 | The Journal of Basic and Applied Zoology

Fig. 5

From: Ameliorative effect of bee venom and its extracted bradykinin-potentiating factor on neurological alteration induced by acrylamide and chips administration

Fig. 5

Photomicrographs of immunohistochemistry of α-synuclein in the CA1 region of the hippocampus of the control group (a). The acrylamide-administrated groups showed a gradual increase in the expression from 30 to 45 days of treatment (arrows; b, c) and denser expression at 60 days (arrows, d). The chips feeding group at 60 days showed increase of intracellular expression (arrows, e). The BV- and BPF-treated groups showed nearly the same expression of the control (f, g). The acrylamide-administrated groups treated with either BV (h) or BPF (i) showed normal expression of α-synuclein similar to the control. The chips feeding groups treated with either BV (j) or BPF (k) showed the normal distribution of staining similar to the control. Immunostaining of α-synuclein; scale bar 5 μm

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