Fig. 9From: Ameliorative effect of bee venom and its extracted bradykinin-potentiating factor on neurological alteration induced by acrylamide and chips administrationPhotomicrographs of immunohistochemistry of α-synuclein in neurons and the glia of the cerebral cortex of the control group (a). The acrylamide-administrated groups showed increase in expression at 30 and 45 days and more denser at 60 days of administration (arrows, b–d), respectively. The chips feeding group (arrows, e) showed increased expression of α-synuclein. The BV- and BPF-treated groups (f, g) at 60 days showed α-synuclein expression similar to the control. The acrylamide-administrated groups treated with either BV (h) or BPF (i) showing downregulated expression of α-synuclein compared to acrylamide administration. The chips feeding groups treated with either BV (j) or BPF (k) showing the normal expression and distribution of staining that was mostly similar to the control. Immunostaining of α-synuclein; scale bar 5 μmBack to article page