Fig. 7From: Ameliorative effect of bee venom and its extracted bradykinin-potentiating factor on neurological alteration induced by acrylamide and chips administrationPhotomicrographs of immunohistochemistry of α-synuclein in the cerebellar region of the control group (a). The acrylamide-administrated groups showed a gradual increase in expression from 30 days (arrows, b) and 45 days (arrows, c) and denser in 60 days of administration (arrows, d). The chips feeding group showed increase of intracellular expression (arrows, e). The BV- and BPF-treated groups showed nearly the same expression of the control (f, g). The acrylamide-administrated groups treated with either BV (h) or BPF (i) showing downregulated expression of α-synuclein expression compared to acrylamide-induced overexpression. The chips feeding groups treated with either BV (j) or BPF (k) showed the normal expression and distribution of staining similar to the control. Immunostaining of α-synuclein; scale bar 5 μmBack to article page